What is jaundice 125 and how to deal with it
Generally speaking, if jaundice is 125, this is a normal situation, we know that for infants and young children, once the jaundice is too high, it will often lead to a more serious situation, especially the performance of jaundice in newborns, most of the jaundice of newborns is physiological jaundice, at this time there is no need for treatment, as long as the conditioning can be strengthened, but if it is pathological jaundice, it must be treated by formal methods.
Jaundice 125
125 divided by 27, so that's normal. The jaundice value of children with different gestational ages, different days, and different diseases is different, that is to say, the normal value of jaundice is different every day after each child is born, and it needs to be confirmed by looking at the child's condition, but if it is based on the general conventional unit, it should be within the normal range
Causes of neonatal jaundice
When the baby is still a fetus, relying on the placenta to supply blood and oxygen, separated from the mother, the baby begins to use its own lungs to breathe, obtain oxygen, the low-oxygen environment in the body changes, no longer need so many red blood cells, then bilirubin is produced, and may be excessive. Excess bilirubin cannot be excreted from the body through an immature enzyme system, and follows the blood flow to all parts of the baby's body, reflecting external signs, that is, the baby's skin and sclera turning yellow. This phenomenon is called neonatal jaundice.
1. Bilirubin is produced
(1) Red blood cell destruction: the fetus is in a low oxygen environment in the mother's womb, and the red blood cells are compensatorily increased, but the life span is short, the blood oxygen content increases after birth, and the excessive red blood cells are quickly destroyed.
(2) High heme oxygenase content: the content is high within 7 days after birth, and the potential for producing bilirubin is high.
2. Immature liver function
(1) Poor ability of liver to take bilirubin: the content of Y and Z proteins in liver cells is insufficient, so that the liver does not take up bilirubin enough.
(2) Poor function of hepatic conjugated bilirubin: the content of glucuronosyltransferase in the liver is low and the activity is insufficient, and the function of conjugated bilirubin is poor.
(3) Poor function of hepatic excretion of bilirubin: the function of excretion of conjugated bilirubin is poor, which is easy to cause cholestasis.
3. Characteristics of entero-liver circulation: the normal flora in the intestine has not yet been established when the newborn is born, and the bilirubin entering the intestine cannot be converted into urobilinogen (fecocobilinogen).
Due to the above characteristics, the ability of newborns to ingest, bind, and excrete bilirubin is obviously not as good as that of adults, and bilirubin is produced more and excreted less, so jaundice is easy to occur. In particular, jaundice will be aggravated when the baby is hypoxic, delayed meconium discharge, delayed feeding, vomiting, dehydration, acidosis, and cerebral hematoma.